Fibrosis-Inflammation Lab
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Fibrosis-Inflammation Lab

Accelerating fibrosis and inflammation research through validated preclinical models and expert insights.

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Fibrosis Pathway Map

Explore key fibrosis signaling pathways and their connected models and drugs.

TGF-βTGF-β ReceptorSmad2/3Nucleus (Transcription)Collagen Production
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Pathway Deep-Dive Articles

In-depth articles on molecular mechanisms, roles in fibrosis, and drug discovery strategies for each signaling pathway.

NF-κB: Stopping Inflammation Without Fueling Fibrosis

NF-κB drives inflammation, but systemic blockade cripples host defense. Dual role in MASH, UUO, IPF models and selective IKK strategies reviewed.

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YAP/TAZ and Tissue Stiffness: Mechanotransduction Targets

YAP/TAZ mechanosensors drive a stiffness-fibrosis feedback loop. Learn the Hippo pathway biology and how PCLS models target it.

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HIF Pathway & Fibrosis: Hypoxia, CKD/MASH/IPF & HIF-PHI

HIF-1α/2α master hypoxic transcription in CKD, MASH, and IPF fibrogenesis. Covers VHL-PHD regulation, organ evidence, and HIF-PHI drugs (Roxadustat).

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ISR in Fibrosis: eIF2α-ATF4 Pathway to ISRIB Drug Discovery

Four kinases (PERK/PKR/GCN2/HRI) converge on eIF2α-ATF4. ISR role in IPF, MASH, and neurodegeneration, plus the ISRIB-led drug development landscape.

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Why TGF-β Inhibitors Fail: Smad Pathway Strategies

TGF-β, the fibrosis master switch, is hard to drug (immunosuppression/cancer). Non-canonical Smad and integrin inhibitors enable selective blockade.

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Wnt/β-catenin: Why a Developmental Pathway Drives Fibrosis

The fetal developmental program, reactivated in adults, can trigger fibrosis. We explore TGF-β crosstalk and Wnt pathway potential as a drug target.

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